Physical activity in case of hormonal failure

You can often hear the question whether it makes sense to train for hormonal failure. Apparently, it occurs in connection with the belief that the hormones circulating in the body completely determine the changes that occur with our body. Whether it is an increase in muscle mass or a decrease in fat reserves.

However, this belief is false, highly hypertrophied. Of course, hormones are the strongest regulators of metabolic processes, but we also have other mechanisms that allow us to respond adequately to the stimuli created by physical activity.

The role negormonalny activation mechanisms of enzyme reactions

Any processes that occur in cells, tissues, or organs occur exclusively under the action of the corresponding enzymes. The more active the enzymes, the more pronounced the reaction. The action of hormones is just due to their activation of enzymes. So active physical activity can itself have a similar effect. For example, in order for glucose from the blood to enter the muscle cells, undergo oxidation-conversion into energy and store up as glycogen, the muscle cell must receive a signal from the hormone insulin. It activates enzymes that provide glucose transport, glycolysis and glycogenesis. And in order for the muscle cell to oxidize fat more than glucose, it must receive a signal from the hormone somatropin.

So, after physical activity, mainly anaerobic orientation (training with weights), all of the above reactions are activated independently of hormonal signals. Glucose circulating in the bloodstream is utilized by cells even without the participation of insulin, as, for example, brain cells do, and fat oxidation increases without increasing the level of growth hormone.

It is noteworthy that the first 3-4 hours after training, insulin, which has properties to inhibit the oxidation of fat in cells, can not implement this function. Fats continue to be oxidized in an increased amount, no matter how much the concentration of insulin in the blood changes (which occurs in response to carbohydrates and proteins consumed). Access to fat reserves, their mobilization from the depot is regulated by a whole set of hormones-epinephrine, norepinephrine, somatropin, cortisol, glucagon, which, if necessary, can compensate for insufficient secretion of each other or excessive secretion of hormones with the opposite effect, the same insulin. In the case of hormonal failures in the endocrine system, access to fat reserves never stops as long as there is a lack of energy and the load is one of the tools for creating it.

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